+91 80860 00608 [email protected] Open All Days · 7 Clinics
Home
All Conditions Acne Psoriasis Eczema Vitiligo Melasma Alopecia Dandruff Ringworm Rosacea Fungal Infection Seborrheic Dermatitis Acne Scars Hyperpigmentation Warts Contact Dermatitis Keloids Urticaria (Hives) Lichen Planus Folliculitis Skin Tags
Hair Transplant Platelet Rich Plasma (PRP) GFC PRP Platelet-Rich Fibrin (PRF) Mesotherapy Beard & Mustache Transplant Eyebrow Transplantation Medical Treatments for Hair Loss
HydraFacial & OxyGeneo Facial Skin Peels MNRF Laser Toning Carbon Laser Peel Botox Fillers & Volume Loss Thread Lift Skin Booster Injections Non-Surgical Rhinoplasty Non-Surgical Jawline & Chin Laser Hair Reduction Laser Tattoo Removal Fractional CO2 Lasers Surgical CO2 Ablation Surgical Tattoo Excision Mole, Wart & Skin Tag Removal Underarm Lightening Hand Rejuvenation Excessive Sweating Body Peel Hand and Foot Peel
Eye Bag Reduction Liposuction Gynecomastia Arm Fat Correction Ear Reshaping Surgery Cyst Excision Double Chin Reduction Fat Grafting
GLP-1 Injections Semaglutide (Ozempic & Wegovy) Tirzepatide (Mounjaro) Belly Fat Reduction Clinical Nutrition Plan Fitness & Mobility Protocol
Gallery
Coimbatore Thrissur Kochi Aluva Kottayam Thiruvalla Thiruvananthapuram Kollam
BOOK A SESSION
📞 +91 80860 00608 [email protected]
Inflammatory Physician Reviewed

Lichen Planus — managed by dermatologists, not masked by creams

A chronic autoimmune inflammatory condition affecting skin, mouth, nails, and scalp — diagnosed accurately and treated with immunomodulatory precision at DermaVue's 7 clinics across Kerala & Tamil Nadu.

LP Oral Lichen Planus Cutaneous Lichen Planus Lichen Planus Pigmentosus
Affects Skin, Mouth, Nails, Scalp
Age Group 30 – 60 years
Contagious No
Treatment 4 – 8 sessions
Consultation ₹300
At a Glance
0–2%
of the global population affected — higher prevalence reported in Indian studies
0×
more common: LP pigmentosus variant in Fitzpatrick IV–VI (Indian/South Asian skin)
0%+
DermaVue patient satisfaction across 7,400+ reviews
Book Consultation WhatsApp for Appointment Find Nearest Clinic →

7 clinics · Kerala & Tamil Nadu · ₹300 consultation

What Is It

Understanding Lichen Planus

Reviewed by Dr. Minu Liz Mathew, MBBS MD DVL — February 2026

Lichen planus is a condition where your immune system mistakenly attacks the skin, mouth lining, nails, or scalp — causing itchy purple bumps, painful mouth sores, or nail damage. It is not caused by infection and is absolutely not contagious. In Indian skin, lichen planus frequently leaves behind stubborn dark patches (post-inflammatory hyperpigmentation) that persist long after the active disease resolves. The good news: lichen planus is very treatable. DermaVue dermatologists use a combination of potent topical steroids, phototherapy, and systemic immunomodulators to control flares, relieve symptoms, and prevent pigmentary complications — all tailored to your skin type and disease pattern.

Lichen planus is a T-cell–mediated autoimmune inflammatory dermatosis characterised by the "5 Ps": pruritic, polygonal, planar (flat-topped), purple papules and plaques. Histopathologically, it demonstrates a band-like (lichenoid) lymphocytic infiltrate at the dermoepidermal junction with Civatte bodies (apoptotic keratinocytes), irregular acanthosis (sawtooth pattern), wedge-shaped hypergranulosis, and Max Joseph spaces. Wickham striae — fine white reticular lines visible on dermoscopy — are pathognomonic. The condition affects cutaneous surfaces, oral mucosa (40–70% of patients), nails (10%), and scalp (lichen planopilaris). Lichen planus pigmentosus (LPP), a variant disproportionately common in Fitzpatrick IV–VI skin, presents with diffuse hyperpigmented macules without preceding inflammatory papules — a major concern in the Indian dermatology population.[1]

M
Dr. Minu Liz Mathew, MBBS MD DVL
Consultant Dermatologist · RealSelf Recognised · DermaVue Kochi
Last reviewed: February 2026
Signs & Symptoms

What does Lichen Planus look like?

Symptoms range widely in severity. Identifying which type you have determines the right treatment.

Purple Flat-Topped Papules
Shiny, violaceous (purple-red) polygonal papules — the hallmark of cutaneous lichen planus. Typically appear on wrists, ankles, lower back, and shins.
Moderate
Wickham Striae
Fine white lace-like lines visible on the surface of papules — pathognomonic for lichen planus. Best seen with dermoscopy or after applying oil.
Moderate
Intense Itching (Pruritus)
Moderate to severe itching that worsens at night. Scratching can trigger new lesions along the scratch line (Koebner phenomenon).
Mod. Severe
Oral Erosions & White Patches
Lace-like white patches (reticular) on inner cheeks, or painful erosions and ulcers on gums, tongue, and palate. Affects 40–70% of LP patients.
Mod. Severe
Nail Changes
Longitudinal ridging, thinning, splitting, or complete nail destruction (pterygium). Affects ~10% of LP patients. Can be permanent if untreated.
Moderate
Post-Inflammatory Pigmentation
Dark brown to grey-brown patches persisting months to years after active lesions resolve. Extremely common and distressing in Indian skin types.
Mod. Severe
Scalp Involvement (Lichen Planopilaris)
Scarring alopecia with perifollicular scaling and erythema. If untreated, causes permanent hair loss in affected areas.
Severe
Genital Lesions
Erosive or papular lesions on genital mucosa causing pain, burning, or discomfort. Often underreported. Requires specialist evaluation.
Mod. Severe
Root Causes

What actually causes Lichen Planus?

Multiple factors act together — understanding them helps prevent recurrence after treatment.

🛡️
Autoimmune T-Cell Attack
CD8+ cytotoxic T lymphocytes mistakenly target basal keratinocytes at the dermoepidermal junction, causing apoptosis and the characteristic lichenoid inflammation. The exact trigger remains unknown in most cases.
🦠
Hepatitis C Association
Strong epidemiological link — hepatitis C virus infection is found in 10–35% of LP patients in endemic populations. HCV screening is recommended for all newly diagnosed LP patients. The association is strongest with oral and erosive LP.
Stress & Psychological Triggers
Acute psychological stress and chronic anxiety are well-documented triggers for LP flares. Stress-induced cortisol dysregulation may alter T-cell–mediated immune responses. Many patients report onset following major life stressors.
💊
Drug-Induced (Lichenoid Eruption)
ACE inhibitors, beta-blockers, NSAIDs, antimalarials, thiazide diuretics, and gold salts can trigger lichenoid drug eruptions clinically indistinguishable from idiopathic LP. Full medication review is essential at diagnosis.
🦷
Dental Materials & Oral Triggers
Amalgam fillings, dental composites, and metal restorations can trigger oral LP through contact hypersensitivity. Replacement with biocompatible materials often improves oral lesions in sensitised individuals.
🧬
Genetic Susceptibility
HLA associations (HLA-DR1, HLA-B7) suggest genetic predisposition. Family clustering is reported in 1–2% of cases. Indian populations show higher LP pigmentosus prevalence, likely reflecting Fitzpatrick phototype–linked genetic factors.
Who gets lichen planus in India?
  • 1–2% of the global population is affected — with Indian studies reporting higher prevalence rates
  • Adults aged 30–60 are most commonly affected; uncommon in children under 10
  • Women are more frequently affected by oral lichen planus, with a female:male ratio of approximately 1.5:1
  • Lichen planus pigmentosus is significantly more common in South Asian, Middle Eastern, and darker-skinned populations (Fitzpatrick IV–VI)
  • Hepatitis C prevalence in LP patients in India ranges from 5–15%, warranting routine screening at diagnosis
Diagnosis Process

What happens at your DermaVue consultation?

A structured clinical assessment — not a quick glance and a prescription pad. Here's exactly what to expect.

01
Clinical Examination & Dermoscopy
Dermatologist identifies characteristic morphology — purple polygonal papules with Wickham striae on dermoscopy. Distribution pattern (flexural, mucosal, nail) documented. Baseline photographs taken.
02
Oral & Mucosal Assessment
Systematic oral cavity examination for reticular white patches, erosions, or desquamative gingivitis. Genital mucosal surfaces examined. Oral LP often coexists with cutaneous LP and may be the only manifestation.
03
Skin Biopsy & Histopathology
Punch biopsy confirms diagnosis — demonstrating band-like lichenoid infiltrate, Civatte bodies, sawtooth acanthosis, and hypergranulosis. Direct immunofluorescence (DIF) may show fibrinogen deposits at the basement membrane zone.
04
Hepatitis C & Lab Screening
Anti-HCV antibody testing recommended for all LP patients per IADVL guidelines. Liver function tests, CBC, and autoimmune markers ordered based on clinical presentation and systemic symptoms.
05
Personalised Treatment Plan
A written protocol combining potent topical corticosteroids, calcineurin inhibitors, phototherapy, or systemic immunomodulation — tailored to disease subtype (cutaneous, oral, nail, pigmentosus), severity, and Indian skin type.
Available at DermaVue

Lichen Planus treatments we offer

All procedures by board-certified MD DVL dermatologists. US-FDA approved equipment. No technician-only protocols — ever.

Find Lichen Planus Treatment Near You
Treatment Journey

Your Lichen Planus treatment timeline

Results are gradual, progressive, and lasting with the right protocol.

Week 1
Consultation, clinical grading, dermoscopy, and baseline photos. Biopsy if needed. Hepatitis C screening ordered. Medication review completed.
Potent topical corticosteroid started. Oral antihistamine for pruritus. Oral LP patients begin tacrolimus ointment.
Month 1
Active itching reduces significantly. Papules begin flattening. Oral erosions show early healing with topical calcineurin inhibitors.
Phototherapy course initiated for generalised LP (2–3 sessions/week). Hepatitis C results reviewed and managed if positive.
Month 2–3
50–70% papule resolution. Oral ulcers healing. Residual hyperpigmentation becoming the dominant concern in Indian skin.
Topical depigmenting agents introduced. Chemical peels started for pigmentation. Steroid taper begins for controlled disease.
Month 4–6
Active disease largely controlled. Post-inflammatory pigmentation fading with peels and topicals. Maintenance protocol established.
Phototherapy sessions reduced to once weekly then stopped. Calcineurin inhibitors continued for oral LP maintenance.
Month 6+
Sustained remission in most patients. Pigmentation continues improving. Quarterly monitoring recommended — LP can relapse.
Flare management plan in place. Most cutaneous LP resolves within 1–2 years; oral LP may require longer maintenance.
FAQ

Frequently asked questions about Lichen Planus

No, lichen planus is absolutely not contagious. It cannot spread from person to person through touch, saliva, or any form of contact. LP is an autoimmune condition where your own immune system attacks skin cells — it is not caused by any infection that can be transmitted. You cannot "catch" lichen planus from someone who has it.

Oral lichen planus affects the mucous membranes inside the mouth — appearing as white lace-like patches (reticular type) or painful erosions and ulcers (erosive type). It affects 40–70% of LP patients and can be the only manifestation. Erosive oral LP causes significant pain while eating and speaking. While malignant transformation risk is low (estimated 0.5–2% over 5 years), regular monitoring every 6 months is recommended by dermatology guidelines. DermaVue dermatologists manage oral LP with topical tacrolimus, steroid mouth rinses, and systemic therapy when needed.

DermaVue consultation fee is ₹300 at most branches. Topical prescription costs vary by medication and area involved. NB-UVB phototherapy sessions range ₹800–1,500 per session (typically 20–30 sessions needed for generalised LP). Chemical peels for residual pigmentation start at ₹1,500–3,500 per session. Full treatment costs are discussed transparently at your first consultation — no hidden charges.

Cutaneous lichen planus is usually self-limiting — most cases resolve within 1–2 years with treatment, though post-inflammatory pigmentation may persist longer. However, oral lichen planus tends to be chronic and may require long-term maintenance therapy. Nail LP and lichen planopilaris (scalp) need early aggressive treatment to prevent permanent nail damage or scarring hair loss. Recurrences can occur, so having a dermatologist-supervised flare plan is important.

Indian skin (Fitzpatrick types IV–VI) has higher melanocyte density and reactivity. When LP causes inflammation at the dermoepidermal junction, melanin drops into the dermis — a process called pigmentary incontinence. This creates persistent grey-brown patches that can last months to years after active LP resolves. Lichen planus pigmentosus (LPP), a variant especially common in South Asians, causes diffuse hyperpigmentation even without obvious preceding papules. DermaVue treats this with chemical peels, topical depigmenting agents, and glutathione therapy tailored to Indian skin.

Yes. Multiple studies — including Indian data — show a statistically significant association between hepatitis C virus (HCV) infection and lichen planus. HCV is found in 5–35% of LP patients depending on the population studied. IADVL guidelines recommend HCV screening for all newly diagnosed LP patients. If positive, treating the underlying hepatitis C often improves LP as well. DermaVue routinely includes HCV screening in the LP diagnostic workup.

Book Today

Stop managing Lichen Planus.
Start clearing it.

Board-certified MD DVL dermatologists across 7 clinics in Kerala & Tamil Nadu. WhatsApp for instant appointment. Consultation ₹300.

Book Consultation WhatsApp Us Find My Nearest Clinic

₹300 consultation · No hidden charges · 7 locations

Book Your Visit

Schedule a Consultation

Board-certified dermatologists across 7 clinics in Kerala & Tamil Nadu.

Scroll to Top

Book a Consultation